Search results for "Macrophage Migration-Inhibitory Factor"

showing 9 items of 9 documents

Macrophage migration inhibitory factor is critically involved in basal and fluoxetine-stimulated adult hippocampal cell proliferation and in anxiety,…

2011

Intensive research is devoted to unravel the neurobiological mechanisms mediating adult hippocampal neurogenesis, its regulation by antidepressants, and its behavioral consequences. Macrophage migration inhibitory factor (MIF) is a pro-inflammatory cytokine that is expressed in the CNS, where its function is unknown. Here, we show, for the first time, the relevance of MIF expression for adult hippocampal neurogenesis. We identify MIF expression in neurogenic cells (in stem cells, cells undergoing proliferation, and in newly proliferated cells undergoing maturation) in the subgranular zone of the rodent dentate gyrus. A causal function for MIF in cell proliferation was shown using genetic (M…

Receptors SteroidStem-Cellsanimal diseasesmedicine.medical_treatmentHippocampusExpressionHippocampal formationHippocampusSubgranular zonememoryMice0302 clinical medicineConditioning PsychologicalCyclin D2Rat Dentate GyrusMice KnockoutNeurons0303 health sciencesMicroscopy ConfocalChronic StressMifNeurogenesisBrainFearrespiratory systemanxietyPsychiatry and Mental healthC-Reactive ProteinCytokinemedicine.anatomical_structuredepressionAntidepressive Agents Second-GenerationStem cellPsychologyAnimal-ModelNeurogenesisSpatial BehaviorNerve Tissue Proteinschemical and pharmacologic phenomena03 medical and health sciencesCellular and Molecular Neurosciencemedicineotorhinolaryngologic diseasesAnimalsRats WistarMaze LearningMacrophage Migration-Inhibitory FactorsMolecular BiologyCell Proliferation030304 developmental biologyMemory DisordersDentate gyrusfluoxetineFactor Mifbiological factorsRatsDisease Models AnimalAcoustic StimulationBromodeoxyuridineMacrophage migration inhibitory factorCorticosteroneNeuroscience030217 neurology & neurosurgery
researchProduct

Spontaneous lymphokine synthesis by human blood mononuclear cells

1975

LYMPHOCYTES, after antigenic stimulation, may synthesise and release biologically active soluble factors other than antibodies. These mediators were termed lymphokines by Dumonde1, and the most extensively studied and best characterised are migration inhibitory factors which can inhibit the migration of macrophages or leukocytes: this is the property used for their in vitro bioassay. Apart from antigens, various other stimuli may trigger lymphokine synthesis by lymphocytes, for example, polyclonal mitogens2, anti-immunoglobulin or membrane Fc or C3-receptor reactions3,4. Furthermore, migration inhibitory activity has been found in the long term culture supernatants of some established lymph…

CellPeripheral blood mononuclear cellMonocytesAntigenmedicineHumansLymphocytesMacrophage Migration-Inhibitory FactorsLymphokinesMultidisciplinarybiologyChemistryLymphokineBiological activityIn vitroCell biologyCold TemperatureBloodmedicine.anatomical_structurePolyclonal antibodiesDepression ChemicalProtein BiosynthesisImmunologybiology.proteinPuromycinAntibodyNature
researchProduct

Roflumilast N-oxide reverses corticosteroid resistance in neutrophils from patients with chronic obstructive pulmonary disease

2013

Background Glucocorticoid functions are markedly impaired in patients with chronic obstructive pulmonary disease (COPD). The phosphodiesterase 4 inhibitor roflumilast N-oxide (RNO) is the active metabolite of roflumilast approved as a treatment to reduce the risk of exacerbations in patients with severe COPD. Objective We sought to characterize the differential effects of RNO versus corticosteroids and their potential additive/synergistic effect in neutrophils from patients with COPD, thus providing scientific rationale for the combination of roflumilast with corticosteroids in the clinic. Methods Peripheral blood neutrophils were isolated from patients with COPD (n = 32), smokers (n = 7), …

CyclopropanesLipopolysaccharidesMaleMAPK/ERK pathwaymedicine.medical_specialtyNeutrophilsPrimary Cell CultureImmunologyDrug ResistanceAminopyridinesGene ExpressionComplex MixturesDexamethasoneHistone DeacetylasesPhosphatidylinositol 3-KinasesPulmonary Disease Chronic ObstructiveGlucocorticoid receptorAdrenal Cortex HormonesInternal medicineTobaccomedicineHumansImmunology and AllergyMacrophage Migration-Inhibitory FactorsDexamethasoneActive metaboliteRoflumilastAgedCOPDbusiness.industryInterleukin-8Drug SynergismMiddle Agedmedicine.diseaseIntramolecular OxidoreductasesEndocrinologyMatrix Metalloproteinase 9BenzamidesMitogen-Activated Protein Kinase PhosphatasesFemaleMacrophage migration inhibitory factorPhosphodiesterase 4 InhibitorsbusinessBiomarkersGlucocorticoidmedicine.drugJournal of Allergy and Clinical Immunology
researchProduct

Cytokine components and mucosal immunity in the oviduct of Xenopus laevis (amphibia, pipidae)

2011

Abstract Most studies on the mucosal immunity in female reproductive tissues have been performed in mammals. In all species, apart from their reproductive strategies, immunity in the genital mucosa is required to defend the host against luminal pathogens. In this study we investigated the role of the innate immunity of the oviductal mucosa of Xenopus laevis , an amphibian characterized by external fertilization. In particular we examined the expression and localization of Interleukin-1β (IL1B), Macrophage migration inhibitory factor (MIF) and Interleukin-1 receptor type 1 (IL1R1) in different oviductal portions including an upper glandular region, an intermediate and a lower aglandular regi…

medicine.medical_treatmentImmunologyInterleukin-1betaXenopus laeviXenopusOviductsXenopus laevisEndocrinologyImmunitymedicineAnimalsImmunity MucosalMacrophage Migration-Inhibitory FactorsReceptors Interleukin-1 Type IInnate immune systembiologybiology.organism_classificationImmunohistochemistryMolecular biologyEpitheliumFemale reproductive tissues; Interleukin-1; Interleukin-1 receptor type 1; Macrophage migration inhibitory factor Xenopus laevismedicine.anatomical_structureCytokineCytokinesOviductImmunohistochemistryFemaleAnimal Science and ZoologyMacrophage migration inhibitory factorGeneral and Comparative Endocrinology
researchProduct

MIF from mussel: coding sequence, phylogeny, polymorphism, 3D model and regulation of expression.

2012

Abstract Three macrophage migration inhibitory factor (MIF)-related sequences were identified from a Mytilus galloprovincialis EST library. The consensus sequence included a 5′-UTR of 32 nucleotides, the complete ORF of 345 nucleotides, and a 3′-UTR of 349 nucleotides. As for other MIFs, M. galloprovincialis ORF does not include any signal or C-terminus extensions. The translated sequence of 115 amino acids possesses a molecular mass of 12,681.4, a pI of 6.27 and a stability index of 21.48. Its 3D structure resembles human MIF except for one shorter α-helix. Although evolutionary separated from ticks and vertebrates, Mg-MIF appeared to be closely related to Pinctada fucata and Haliotis, but…

Cytokine Gene regulation Challenge Bacteria Fungus q-PCR Innate immunity MytiluModels MolecularHemocytesImmunologyMolecular Sequence DataPhylogeneticsConsensus sequenceCoding regionAnimalsPinctada fucataNucleotideAmino Acid SequenceCloning MolecularMacrophage Migration-Inhibitory FactorsPhylogenyGene LibraryGeneticsRegulation of gene expressionchemistry.chemical_classificationExpressed Sequence TagsMytilusbiologyBase SequenceGene Expression Profilingbiology.organism_classificationAmino acidchemistryItalyMacrophage migration inhibitory factorFranceSequence AlignmentDevelopmental BiologyDevelopmental and comparative immunology
researchProduct

A macrophage-suppressing 40-kD protein in a case of pulmonary alveolar proteinosis.

1987

Pulmonary alveolar proteinosis (PAP) is a rare disease of unknown etiology. Macrophage dysfunctions are claimed to be involved in the pathogenesis. We investigated phagocytosis and oxidative metabolism of alveolar macrophages in a case of pulmonary alveolar proteinosis. These cells phagocytize normally and phagocytizable stimulants cause a normal oxidative burst. In response to the membrane signals phorbolmyristate acetate and aggregated immunoglobulin, however, no stimulated turnover of the oxidative metabolism can be observed. A 40-kD protein found in the lavage fluid mediates this macrophage-inhibiting effect. This phenomenon may contribute to the frequent opportunistic infections seen i…

AdultMalePathologymedicine.medical_specialtyPhagocytosisOpportunistic InfectionsPulmonary Alveolar ProteinosisPathogenesisPhagocytosisDrug DiscoverymedicineMacrophageHumansMacrophage Migration-Inhibitory FactorsGenetics (clinical)Lungmedicine.diagnostic_testbiologyMacrophagesfood and beveragesProteinsGeneral MedicineMacrophage Activationmedicine.diseaseRespiratory burstMolecular WeightPulmonary AlveoliBronchoalveolar lavagemedicine.anatomical_structureImmunologyLuminescent Measurementsbiology.proteinMolecular MedicineAntibodyPulmonary alveolar proteinosisEnergy MetabolismBronchoalveolar Lavage FluidKlinische Wochenschrift
researchProduct

Macrophage Migration Inhibitory Factor Induces Inflammation and Predicts Spinal Progression in Ankylosing Spondylitis

2017

Objective: To investigate the role of macrophage migration inhibitory factor (MIF) in the pathogenesis of ankylosing spondylitis (AS). Methods: Patients who met the modified New York criteria for AS were recruited for the study. Healthy volunteers, rheumatoid arthritis patients, and osteoarthritis patients were included as controls. Based on the annual rate of increase in modified Stoke AS Spine Score (mSASSS), AS patients were classified as progressors or nonprogressors. MIF levels in serum and synovial fluid were quantitated by enzyme-linked immunosorbent assay. Predictors of AS progression were evaluated using logistic regression analysis. Immunohistochemical analysis of ileal tissue was…

AnkylosingAdultMaleLogistic ModelMacrophageImmunologyEnzyme-Linked Immunosorbent AssayIntramolecular OxidoreductasePredictive Value of TestMonocyteSeverity of Illness IndexCalcificationCalcification PhysiologicPaneth CellRheumatologySynovial Fluidotorhinolaryngologic diseasesImmunology and AllergySpondylitis AnkylosingPhysiologicSpondylitiMacrophage Migration-Inhibitory FactorTumor Necrosis Factor-alphaOsteoblastB-LymphocyteHistocompatibility Antigens Class IIMiddle AgedSpineAntigens Differentiation B-LymphocyteSettore MED/16 - ReumatologiaAntigenDifferentiationDisease ProgressionFemaleCase-Control StudieHuman
researchProduct

Identification of CPE and GAIT elements in 3’UTR of macrophage migration inhibitory factor (MIF) involved in inflammatory response induced by LPS in …

2018

Innate immune responses face infectious microorganisms by inducing inflammatory responses. Multiple genes within distinct functional categories are coordinately and temporally regulated by transcriptional 'on' and 'off' switches that account for the specificity of gene expression in response to external stimuli. Mechanisms that control transcriptional and post-transcriptional regulation are important in coordinating the initiation and resolution of inflammation. Macrophage migration inhibitory factor (MIF) is an important cytokine that, in Ciona robusta, is related to inflammatory response. It is well known that in C. robusta, formerly known as Ciona intestinalis, the pharynx is involved in…

Lipopolysaccharides0301 basic medicineUntranslated regionImmunology03 medical and health sciences0302 clinical medicineGene expressionAnimalsCiona intestinalisAmino Acid SequenceRNA Processing Post-Transcriptional3' Untranslated RegionsMacrophage Migration-Inhibitory FactorsMolecular BiologyGenePhylogenyInflammationRegulation of gene expressionInnate immune systemBase SequencebiologyThree prime untranslated regionbiology.organism_classificationImmunity InnateCiona intestinalisUp-RegulationAscidianMacrophage migration inhibitory factorInflammationLPSCiona robustaCell biology030104 developmental biologyGene Expression Regulation030220 oncology & carcinogenesisMacrophage migration inhibitory factorSequence AlignmentMolecular Immunology
researchProduct

Blockade of MIF-CD74 Signalling on Macrophages and Dendritic Cells Restores the Antitumour Immune Response Against Metastatic Melanoma.

2018

Mounting an effective immune response against cancer requires the activation of innate and adaptive immune cells. Metastatic melanoma is the most aggressive form of skin cancer. While immunotherapies have shown a remarkable success in melanoma treatment, patients develop resistance by mechanisms that include the establishment of an immune suppressive tumor microenvironment. Thus, understanding how metastatic melanoma cells suppress the immune system is vital to develop effective immunotherapies against this disease. In this study, we find that macrophages (MOs) and dendritic cells (DCs) are suppressed in metastatic melanoma and that the Ig-CDR-based peptide C36L1 is able to restore MOs and …

MaleModels MolecularImmunologyMelanoma Experimentalchemical and pharmacologic phenomenaModels Biologicalimmune responseMiceStructure-Activity RelationshipT-Lymphocyte SubsetsAnimalsdendritic cellsNeoplasm MetastasisReceptors ImmunologicMacrophage Migration-Inhibitory FactorsMelanomaOriginal ResearchMacrophagesHistocompatibility Antigens Class IIImmunitypeptide-based immunotherapyAntigens Differentiation B-LymphocyteCD74macrophage migration inhibitory factorPeptidesProtein BindingSignal Transductionmetastatic melanomaFrontiers in immunology
researchProduct